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Причины Тактильной Сверхчувствительности
autism4us wrote in autism4us_ru
"Skin that is sensitive to the touch is annoying, uncomfortable, and frequently unbearable. Skin pain or tenderness is medically called allodynia, a condition in which one feels an exaggerated type of pain or sensitivity even to non-painful stimuli, such as a breeze. There are different types of skin tenderness, including:
Tactile or mechanical allodynia, which is triggered by touch
Static mechanical allodynia, which is caused by light touch or pressure
Dynamic mechanical allodynia, where brushing the skin triggers pain
Thermal allodynia, which is felt with cold or hot stimuli...
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А Специалисты по сенсорике мед. документов не читают? Тут так все нормально выложено...Перевести бы...

Еще одна из причин - Оральный или Вагинальный Кандидоз Oral and Vaginal Thrush)

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перевести можно, но там вроде бы так немного выложено?

Немного, но там самое главное - причины. Уже это многим бы помогло, потому что на это можно тупо сдать анализы. А то мы будем тратить время на десенсибилизацию, когда вопрос весь в демиелинизации или в витамине В.

Edited at 2012-09-03 09:29 am (UTC)

Невропатия и Витамин В1

"Because vitamin B1 is important for the function of the nervous system, one of the symptoms of a deficiency in vitamin B1 is peripheral neruopathy. Peripheral neuropathy can cause numbness, tingling or a burning sensation in your extremities, especially your hands and feet. You also might develop problems walking, leg weakness, pain, vomiting and strange eye movements due to a vitamin B1 deficiency"

Read more: http://www.livestrong.com/article/427843-vitamin-b1-peripheral-neuropathy/#ixzz25OiLXalW

Edited at 2012-09-03 09:32 am (UTC)

Нейропатия и витамин В6

"The classic clinical syndrome for B6 deficiency is a seborrhoeic dermatitis-like eruption, atrophic glossitis with ulceration, angular cheilitis, conjunctivitis, intertrigo, and neurologic symptoms of somnolence, confusion, and neuropathy.[10]

While severe vitamin B6 deficiency results in dermatologic and neurologic changes, less severe cases present with metabolic lesions associated with insufficient activities of the coenzyme pyridoxal phosphate. The most prominent of the lesions is due to impaired tryptophan-niacin conversion. This can be detected based on urinary excretion of xanthurenic acid after an oral tryptophan load. Vitamin B6 deficiency can also result in impaired transsulfuration of methionine to cysteine. The pyridoxal phosphate-dependent transaminases and glycogen phosphorylase provide the vitamin with its role in gluconeogenesis, so deprivation of vitamin B6 results in impaired glucose tolerance.[4]

A deficiency of vitamin B6 alone is relatively uncommon and often occurs in association with other vitamins of the B complex.
Because data on the adverse effects of vitamin B6 are limited, caution may be warranted. !!!Sensory neuropathy has occurred from high intakes of supplemental forms!!!"

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